Braden Kovalovich - Drexel University - Philadelphia, Pennsylvania, United States | LinkedIn
Deficiency of G-protein-coupled bile acid receptor Gpbar1 (TGR5) enhances chemically induced liver carcinogenesis. - Abstract - Europe PMC
Steatosis does not impair liver regeneration after partial hepatectomy | Laboratory Investigation
Steatosis does not impair liver regeneration after partial hepatectomy | Laboratory Investigation
Deficiency of G-protein-coupled bile acid receptor Gpbar1 (TGR5) enhances chemically induced liver carcinogenesis. - Abstract - Europe PMC
Deficiency of G-protein-coupled bile acid receptor Gpbar1 (TGR5) enhances chemically induced liver carcinogenesis. - Abstract - Europe PMC
Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis - ScienceDirect
Bile Acid Regulation of C/EBPβ, CREB, and c-Jun Function, via the Extracellular Signal-Regulated Kinase and c-Jun NH2-Terminal Kinase Pathways, Modulates the Apoptotic Response of Hepatocytes | Molecular and Cellular Biology
Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis - ScienceDirect
The Scranton Journal Fall 1988 - University of Scranton PR Publications and Press Releases - The University of Scranton Digital Collections
Sustained Phosphorylation of Bid Is a Marker for Resistance to Fas-Induced Apoptosis During Chronic Liver Diseases
PMA MEDICAL SPECIALISTS - GASTROENTEROLOGY CENTER - 13 Reviews - 420 W Linfield-Trappe Rd, Limerick, PA - Yelp
Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis - ScienceDirect
Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis - ScienceDirect
Jon Kovalovich - Supervisor - State of Nevada Department of Employment, Training, & Rehabilitation | LinkedIn
Cytokine control of developmental programs in normal hematopoiesis and leukemia | Oncogene
Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis - ScienceDirect
Podiatry Management Online
Sustained Phosphorylation of Bid Is a Marker for Resistance to Fas-Induced Apoptosis During Chronic Liver Diseases
1987 Commencement Program, University Archives, University of Pennsylvania
IL-6 cytoprotection in hyperoxic acute lung injury occurs via PI3K/Akt-mediated Bax phosphorylation. - Abstract - Europe PMC
Kara Kovalovich - Teacher - Hillsborough County Public Schools | LinkedIn
